Humans are all innately biophiliacs, but society has forced us to remove ourselves from nature. Even now, when we have the opportunity to immerse ourselves in the natural world, we still manage to subject ourselves to the toxicity of the built environment we call society. Whether you’re going to the beach, or the forest; using chemical-based products can not only harm the environment you’re in, but also exert toxic effects on your body.
Oxybenzone is a common ingredient of sunscreens, and not only is it toxic to skin cells, it also damages the reef and forest ecosystems where it usually ends up running off into after one bathes in nature (oceans, lakes, rivers, waterfalls, etc). Oxybenzone (among various other benzones), aluminum, polysorbate 20, and other common sunscreen toxins are silent killer of our oceans. Reefs principally, and consequently all other marine life suffer from their acidifying and persistently contaminating effects.
Oxybenzone and other chemicals in sunscreen make the water acidic after they break down into their (also highly toxic) byproducts and hence decimate photosynthetic algae polyps. These single celled algae living symbiotically on the external surfaces of all coral whereby they exchange photosynthesized sugars (energy/food) for structural support are essential for feeding the lower primary consumers in the reef ecosystems, and if they die, seaweeds have nowhere to anchor themselves, fish have nowhere to live, and after a certain point even crabs and sea cucumbers are left to starve.
If we continue to contaminate mother earth’s blood with man-made persistent industrial pollutants, the oceans will be a barren underwater graveyard.
If you’re not sold on throwing away the Coppertone, I should note that all main-brand sunscreens, insect repellants, and outdoor/cosmetic cream/pastes are filled with many other endocrine disruptors, so if vaccine-like neurotoxicity isn’t enough, feminization of males and reproductive issues in females (infertility, irritability, estrogen dominance) is warranted by the use of all main-brand products (which I feel confident in saying is 90% of the products in use today). Not only this, but we must consider, as vaccine manufacturers never do, the concomitant toxicokinetic mechanisms, just as DEET has been clinically shown to significantly enhance the epidermal absorption of oxybenzone. Or how aluminum, polysorbate 80, triclosan, fluoride, and mercury all act synergistically to cross blood-brain-barrier.
We must do our best to preserve the beauty and purity of our mother earth, while also enjoying all that it has to teach and offer us. Go take a walk in the park, meditate on a rock, climb a mountain, swim in the lake or jump in the ocean, and always leave it better than how you found it. Stay true to your nature, and our mother earth and beloved father sky will guide and protect you in every one of your endeavors.
Oxybenzone is a common ingredient of sunscreens, and not only is it toxic to skin cells, it damages the reef and forest ecosystems where it ends up running off when one bathes in nature.
Acute toxicity testing of TiO2-based vs. oxybenzone-based sunscreens on clownfish (Amphiprion ocellaris).
Our data indicate that at an exposure level of 100 mg/L oxybenzone-containing sunscreen had a negative impact on mortality, leading to 25% death by the end of the 97-h testing period. Negative impacts on behavior were even more dramatic for the 100 mg/L oxybenzone-containing sunscreen, with 100% of the animals failing to feed over the first 49 h of testing and 100% of animals demonstrating abnormal swimming behavior over the entire testing period.
Can oxybenzone cause Hirschsprung’s disease?
Testing in human cell lines confirmed that low levels of oxybenzone has the potential to disrupt cell migration and function in a manner similar to what is associated with HSCR. Analysis of human exposure levels to oxybenzone from sunscreen use, under normal conditions, demonstrates that enough chemical can cross into the mother’s blood making it available to the fetus at high enough levels that can indeed inhibit migration of neural crest cells during critical embryonic development.
– Ozonation of the oxybenzone, octinoxate, and octocrylene UV-filters: Reaction kinetics, absorbance characteristics, and transformation products.
– These 4-dimensional maps also confirmed ozone attack mechanisms, namely reaction at phenolate (OXY) and olefin (OMC, OC) groups. Primary transformation products from these reactions were identified for all three UVFs of concern. For OC and OXY, the benzophenone structure is conserved, suggesting that transformation products retain toxicity concerns.
– Dermatological and environmental toxicological impact of the sunscreen ingredient oxybenzone/benzophenone-3.
– In humans, oxybenzone has been reported to produce contact and photocontact allergy reactions, implemented as a possible endocrine disruptor and has been linked to Hirschsprung’s disease. Environmentally, oxybenzone has been shown to produce a variety of toxic reactions in coral and fish ranging from reef bleaching to mortality. Lastly, with the rise in skin cancer rates and the availability of more effective sunscreen actives such as micronized zinc oxide and titanium dioxide, serious doubts about the relative prevention benefit of personal care products containing oxybenzone must be raised and compared with the potential negative health and environmental effects caused by the accumulation of this and other chemicals in the ecosystem.
– Urinary Concentrations of Triclosan, Benzophenone-3, and Bisphenol A in Taiwanese Children and Adolescents.
– The present study showed that urinary TCS concentrations in females were significantly higher than those in males, respectively. In addition, BP-3 was only detected in urine samples of female adolescents. Sex and age were the important factors influencing urinary TCS and BP-3 concentrations.
– Sunscreen bans: Coral reefs and skin cancer.
– Concentration estimates and mechanism studies support an association-direct or indirect (via promotion of viral infection)-of sunscreens with bleaching of coral reefs. A ban on the two most common sunscreen ingredients goes into effect in Hawaii on January 1, 2021. Proponents suggest that this is a trend, just the first of many such bans worldwide; opponents warn of a dire increase in human skin cancers. As a result, alternative sunscreen compounds are being sought.
– Update About the Effects of the Sunscreen Ingredients Oxybenzone and Octinoxate on Humans and the Environment.
– The use of the two most common organic filters, oxybenzone and octinoxate, has recently been restricted in Hawaii due to their harmful effect on the coral reefs. Here, we discuss recent studies about these specific filters related to the adverse health risks they pose for humans and other organisms, as well as environmental repercussions.
– Significant inhibition of photosynthesis and respiration in leaves of Cucumis sativus L. by oxybenzone, an active ingredient in sunscreen.
– The inhibition of photosynthesis also enhanced the excess excitation energy in chloroplasts, resulting in overproduction of reactive oxygen species (ROS), and the inhibition of respiration aggravated this process. ROS accumulation adversely affects the structure and function of proteins, DNA and membrane lipids in plant cells, interfering with normal metabolism and even leading to plant death. Therefore, reducing the use of OBZ is important for protecting global ecological security.
– Mapping the intrinsic absorption properties and photodegradation pathways of the protonated and deprotonated forms of the sunscreen oxybenzone.
– First application of carbon-based screen-printed electrodes for the voltammetric determination of the organic UV filters oxybenzone and octocrylene.
– A new voltammetric methodology is proposed for the simultaneous determination of the organic UV filters oxybenzone (benzophenone 3, BP3) and octocrylene (OC) in personal care products and in wastewater extracts.
– Can oxybenzone cause Hirschsprung’s disease?
– Oxybenzone is a ultraviolet (UV) absorber used in 70% of sunscreen products, is a recognized endocrine disrupting chemical (EDC) and is small enough to pass through skin and placenta barriers. Numerous studies have identified this chemical in the urine/blood of pregnant women as well as in fetal and umbilical cord blood. A recent study demonstrated that women with medium to high levels of oxybenzone in their urine was associated with giving birth to neonates with Hirschsprung’s Disease (HSCR). Testing in human cell lines confirmed that low levels of oxybenzone has the potential to disrupt cell migration and function in a manner similar to what is associated with HSCR. Analysis of human exposure levels to oxybenzone from sunscreen use, under normal conditions, demonstrates that enough chemical can cross into the mother’s blood making it available to the fetus at high enough levels that can indeed inhibit migration of neural crest cells during critical embryonic development.